The binding of heparin to cTnI may decrease immunoreactivity, dependant upon the heparin concentration in sample collection tubes (e.g., heparin in a concentration of ninety U/mL is documented to lead to a lessen of close to 20% in cTnI focus).[33] The effect of heparin on cTnI immunoassays can be induced by changes throughout the sample matrix.
This issue, having said that, is unwarranted and is based on several misconceptions. In truth, newer troponin assays symbolize an important improvement in laboratory screening, and when utilised effectively will improve clinicians' ability to speedily diagnose patients with suspected acute coronary syndromes (ACS). These concerns had been resolved in our modern assessment article "Troponin Testing for Clinicians"4 and they are briefly reviewed here.
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